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Cell Death/Damage: Mechanisms

MitochondrialIon Channels / Membrane
W
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mother/apoptosis.htm#Cell Death/Damage: Mechanisms 9

Overview

From WUSTL Neuromuscular

Cuproptosis Pyroptosis Necroptosis Autophagy Apoptosis Ferroptosis Necrosis Wallerian Degeneration Consequence Cell death Inflammatory cell death Necrosis Self-degradationCell death when excessive Cell death Cell death Cell death Axon degeneration Molecules/Triggers Copper ↑ Inflammasome activation TNF, TLRs StressOrganelle damage Mitochondrial, orExtrinsic Iron ↑GPX4 inhibitionLipid peroxidation Calcium ↑ NMN/NAD+ ↑TNF-αMLKL Mechanisms Cu binding to lipoylated proteinsMitochondrial Δ Caspase-1 cleavagePore formation RIPK3-MLKLMembrane rupture Lysosomal degradation Caspase activationDNA fragme

Related Conditions

Shared genes
Acute Motor (Axonal) Neuropathy (AMAN)
IIGQ1BGM1
Bone Morphogenetic Proteins
II
Vici syndrome
IICLINICAL

External Resources

WUSTL Neuromuscular
Washington University Disease Center
PubMed
Biomedical Literature
GeneReviews
NCBI Expert-Authored Reviews
Orphanet
Portal for Rare Diseases
NORD
National Organization for Rare Disorders

Data sourced from the Washington University Neuromuscular Disease Center. For clinical use, always refer to primary sources.